{"id":9227,"date":"2025-11-22T13:28:17","date_gmt":"2025-11-22T10:28:17","guid":{"rendered":"https:\/\/jordan-cardiac.org\/?p=9227"},"modified":"2025-11-22T13:29:06","modified_gmt":"2025-11-22T10:29:06","slug":"jordan-cardiac-society-jcs-taskforce-congress-october-2025-the-jordanian-task-force-protocol-for-manegement-of-patients-with-wide-complex-tachycardia-1","status":"publish","type":"post","link":"https:\/\/jordan-cardiac.org\/en\/jordan-cardiac-society-jcs-taskforce-congress-october-2025-the-jordanian-task-force-protocol-for-manegement-of-patients-with-wide-complex-tachycardia-1\/","title":{"rendered":"Jordan Cardiac Society (JCS) Taskforce Congress, October 2025  The Jordanian Task Force Protocol for Manegement of patients with Wide Complex Tachycardia [1]"},"content":{"rendered":"<p class=\"s3\"><span class=\"s2\">Jordan Cardiac Society (JCS) Taskforce Congress, October 2025<\/span><\/p>\n<p class=\"s3\"><span class=\"s2\">The Jordanian Task Force Protocol for Manegement of patients with <\/span><span class=\"s2\">W<\/span><span class=\"s2\">ide <\/span><span class=\"s2\">C<\/span><span class=\"s2\">omplex <\/span><span class=\"s2\">T<\/span><span class=\"s2\">achycardia<\/span><span class=\"s2\"> [1]<\/span><\/p>\n<p class=\"s3\"><span class=\"s4\">Laith Saleh, MD and Basil Abu El Haija, MD<\/span><\/p>\n<p class=\"s6\"><span class=\"s5\">Tachycardias are broadly categorized <\/span><span class=\"s5\">into wide complex and narrow complex tachycardia (based on QRS width<\/span><span class=\"s5\">, with a cut off of 120 ms)<\/span><\/p>\n<p class=\"s8\"><span class=\"s7\">Initial management<\/span><\/p>\n<p class=\"s8\"><span class=\"s5\">The provider should conduct a brief history and physical examination with an <\/span><span class=\"s5\">assessment of the symptoms, vital signs, and level of consciousness to determine if they are hemodynamic stable or unstable.<\/span><span class=\"s5\"> During the clinician assessment<\/span><span class=\"s5\">, other members of the health care team should:<\/span><\/p>\n<div class=\"s10\"><span class=\"s9\">\u2022 <\/span><span class=\"s5\">Administer supplemental oxygen<\/span><\/div>\n<div class=\"s10\"><span class=\"s9\">\u2022 <\/span><span class=\"s5\">Establish intravenous access<\/span><\/div>\n<div class=\"s10\"><span class=\"s9\">\u2022 <\/span><span class=\"s5\">Send blood for appropriate initial studies<\/span><\/div>\n<div class=\"s10\"><span class=\"s9\">\u2022 <\/span><span class=\"s5\">Attach the patient to a continuous cardiac monitor<\/span><\/div>\n<div class=\"s10\"><span class=\"s9\">\u2022 <\/span><span class=\"s5\">Obtain a 12-lead ECG<\/span><\/div>\n<p class=\"s8\"><span>\u00a0<\/span><\/p>\n<p class=\"s8\"><span class=\"s11\">Unstable patients<\/span><\/p>\n<div class=\"s10\"><span class=\"s9\">\u2022 <\/span><span class=\"s5\">Unstable<\/span><span class=\"s5\">, <\/span><span class=\"s5\">pulseless: <\/span><span class=\"s5\">Patients with WCT who are pulseless, or who become pulseless during the course of evaluation and treatment, should be managed according to<\/span><span class=\"s5\"> <\/span><span class=\"s5\">standard advance cardiac life support (ACLS) resuscitation algorithms<\/span><\/div>\n<div class=\"s10\"><span class=\"s9\">\u2022 <\/span><span class=\"s5\">Unstable, with a pulse: P<\/span><span class=\"s5\">atients with WCT who are hemodynamically unstable, but still responsive with a discernible blood pressure and pulse<\/span><span class=\"s5\"> should undergo <\/span><span class=\"s5\">urgent<\/span><span class=\"s5\">,<\/span><span class=\"s5\"> <\/span><span class=\"s5\">synchronized when possible, <\/span><span class=\"s5\">cardioversion (with procedural sedation when feasible).<\/span><\/div>\n<p class=\"s8\"><span>\u00a0<\/span><\/p>\n<p class=\"s8\"><span class=\"s11\">Stable patients with uncertain WCT etiology<\/span><\/p>\n<p class=\"s8\"><span class=\"s5\">For hemodynamically stable patients with WCT which is regular and monomorphic in whom the etiology of the WCT remains uncertain, we suggest the following approach:<\/span><\/p>\n<p class=\"s12\"><span>\u00a0<\/span><\/p>\n<div class=\"s14\"><span class=\"s13\">1. <\/span><span class=\"s5\">Perform vagal maneuvers (Valsalva, carotid sinus massage, etc)<\/span><span class=\"s5\">: Valsalva maneuvers, cartoid massage, ice water immertion<\/span><span class=\"s5\">, and assess response:<\/span><\/div>\n<div class=\"s16\"><span class=\"s15\">o <\/span><span class=\"s5\">Sinus tachycardia will gradually slow during the maneuver and then accelerate upon completion of the maneuver.<\/span><\/div>\n<div class=\"s16\"><span class=\"s15\">o <\/span><span class=\"s5\">During atrial tachycardia or atrial flutter, the ventricular response will transiently slow (due to increased AV nodal blockade). The arrhythmia itself, which occurs within the atria, is usually unaffected<\/span><span class=\"s5\"> and often better recognized with a slower ventricular rate<\/span><span class=\"s5\">.<\/span><\/div>\n<div class=\"s16\"><span class=\"s15\">o <\/span><span class=\"s5\">A paroxysmal SVT (either AVNRT or AVRT) will frequently terminate because of the dependence on the AV node.<\/span><\/div>\n<div class=\"s16\"><span class=\"s15\">o <\/span><span class=\"s5\">VT is generally unaffected by vagal maneuvers, but the effect on the AV node may expose AV dissociation by altering the sinus rate (or PP intervals). VT termination due to carotid sinus pressure is rare.<\/span><\/div>\n<div class=\"s14\"><span class=\"s13\">2. <\/span><span class=\"s17\">Administer<\/span><span class=\"s17\"> adenosine<\/span><\/div>\n<p class=\"s18\"><span class=\"s5\">Resuscitation equipment should be immediately available as rarely adenosine will precipitate hemodynamic collapse. The initial dose of adenosine is 6 mg; if this has no effect, 12 mg can be administered.<\/span><\/p>\n<p class=\"s18\"><span class=\"s5\">AVNRT and AVRT will frequently be terminated following adenosine administration. Adenosine will <\/span><span class=\"s5\">also <\/span><span class=\"s5\">terminate some uncommon<\/span><span class=\"s5\"> adenosine-sensitive atrial tachycardia<\/span><span class=\"s5\"> <\/span><span class=\"s5\">and<\/span><span class=\"s5\"> some VTs <\/span><span class=\"s5\">like idiopathic right ventricular outflow tract (RVOT) VT.<\/span><span class=\"s5\"> <\/span><span class=\"s5\">However, adenosine has no effect on most tachycardias that are not AV-node dependent. <\/span><\/p>\n<p class=\"s18\"><span>\u00a0<\/span><\/p>\n<div class=\"s16\"><span class=\"s15\">o <\/span><span class=\"s5\">Adenosine is administered via rapid intravenous push, followed immediately by 10 mL<\/span><span class=\"s5\">\u00a0<\/span><span class=\"s5\">saline<\/span><span class=\"s5\">\u00a0<\/span><span class=\"s5\">flush<\/span><span class=\"s5\">using a proximal canula (AC or above)<\/span><\/div>\n<div class=\"s16\"><span class=\"s15\">o <\/span><span class=\"s5\">If <\/span><span class=\"s5\">adenosine is administered properly as above and <\/span><span class=\"s5\">there is no change in the ventricular rate and rhythm, the WCT is likely <\/span><span class=\"s5\">to be <\/span><span class=\"s5\">VT. <\/span><\/div>\n<div class=\"s16\"><span class=\"s15\">o <\/span><span class=\"s5\">I<\/span><span class=\"s5\">n atrial arrhythmia <\/span><span class=\"s5\">like atrial <\/span><span class=\"s5\">tachycaredia<\/span><span class=\"s5\"> or atrial flutter, adenosine usually slows <\/span><span class=\"s5\">the ventricular <\/span><span class=\"s5\">rate <\/span><span class=\"s5\">temporarily <\/span><span class=\"s5\">and<\/span><span class=\"s5\"> the <\/span><span class=\"s5\">underlying<\/span><span class=\"s5\"> atrial activity is typically easily seen on the ECG<\/span><span class=\"s5\">.<\/span><\/div>\n<div class=\"s14\"><span class=\"s13\">3. <\/span><span class=\"s5\">Avoidance of other pharmacologic agents<\/span><\/div>\n<p class=\"s18\"><span class=\"s5\">Intravenous beta blockers, calcium channel blockers, and<\/span><span class=\"s5\"> digoxin <\/span><span class=\"s5\">are not typically used, due to the potential for hemodynamic deterioration<\/span><\/p>\n<div class=\"s14\"><span class=\"s13\">4. <\/span><span class=\"s5\">Further treatment is directed by the response to vagal maneuvers and\/or<\/span><span class=\"s5\"> adenosine<\/span><span class=\"s5\">, specifically targeting VT or the relevant SVT. If the WCT persists and the etiology remains uncertain, <\/span><span class=\"s5\">the treatment should assume that <\/span><span class=\"s5\">the WCT is VT and treat accordingly.<\/span><\/div>\n<p class=\"s8\"><span>\u00a0<\/span><\/p>\n<p class=\"s12\"><span class=\"s11\">Stable patients with known WCT etiology<\/span><\/p>\n<p class=\"s12\"><span class=\"s5\">In a patient with WCT who is hemodynamically stable, therapy may be targeted to the specific arrhythmia (VT or SVT) when identifiable from the available data.<\/span><\/p>\n<p class=\"s19\"><span class=\"s11\">Ventricular tachycardia<\/span><\/p>\n<div class=\"s20\"><span class=\"s9\">\u2022 <\/span><span class=\"s5\">If electrical cardioversion with appropriate procedural sedation is the chosen approach, intravenous analgesics or sedatives should be cautiously administered if the blood pressure will tolerate their use.<\/span><\/div>\n<div class=\"s20\"><span class=\"s9\">\u2022 <\/span><span class=\"s5\">If p<\/span><span class=\"s5\">harmacologic cardioversion is the chosen approach,<\/span><span class=\"s5\"> <\/span><span class=\"s5\">intravenous<\/span><span class=\"s5\"> amiodarone <\/span><span class=\"s5\">or lidocaine should be used<\/span><\/div>\n<div class=\"s20\"><span class=\"s9\">\u2022 <\/span><span class=\"s5\">Any associated conditions should be treated, including cardiac ischemia, heart failure, electrolyte abnormalities, or drug toxicities.<\/span><\/div>\n<div class=\"s20\"><span class=\"s9\">\u2022 <\/span><span class=\"s5\">For patients with one of the known syndromes of VT in structurally normal hearts, calcium channel blockers or beta blockers may be used, particularly if the patient has been successfully treated in the past with such medications. These drugs can be used either to terminate the arrhythmia, or after cardioversion to suppress recurrences<\/span><span class=\"s5\">.<\/span><\/div>\n<p class=\"s21\"><span>\u00a0<\/span><\/p>\n<p class=\"s21\"><span class=\"s11\">Supraventricular tachycardia<\/span><\/p>\n<p class=\"s22\"><span class=\"s5\">Once the WCT has been definitively established as SVT, therapy directed at the SVT may be given. In such cases, management is similar to an SVT with a normal QRS duration<\/span><span class=\"s5\">, and SVT protocols should be implemented with no significant difference<\/span><span class=\"s5\">.<\/span><\/p>\n<p class=\"s23\"><span>\u00a0<\/span><\/p>\n<p class=\"s24\"><span>\u00a0<\/span><\/p>\n<div class=\"s26\"><span class=\"s25\">1. <\/span><span class=\"s11\">Adapted from<\/span><span class=\"s5\"> <\/span><span class=\"s11\">2019 ESC Guidelines for the management of patients with supraventricular tachycardia. The Task Force for the management of patients with supraventricular tachycardia of the European Society of Cardiology (ESC). <\/span><span class=\"s11\">Eur<\/span><span class=\"s11\"> Heart J 2020; 41:655; 2017 AHA\/ACC\/HRS Guideline for Management of Patients with Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death: A Report of the American College of Cardiology\/American Heart Association Task Force on Clinical Practice Guidelines and the Heart Rhythm Society<\/span><\/div>\n<p class=\"s24\"><span>\u00a0<\/span><\/p>\n<p class=\"s24\"><span>\u00a0<\/span><\/p>\n","protected":false},"excerpt":{"rendered":"<p>Jordan Cardiac Society (JCS) Taskforce Congress, October 2025 The Jordanian Task Force Protocol for Manegement of patients with Wide Complex Tachycardia [1] Laith Saleh, MD and Basil Abu El Haija, MD Tachycardias are broadly categorized into wide complex and narrow complex tachycardia (based on QRS width, with a cut off of 120 ms) Initial management [&hellip;]<\/p>\n","protected":false},"author":145,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[1],"tags":[],"class_list":["post-9227","post","type-post","status-publish","format-standard","hentry","category-uncategorized"],"_links":{"self":[{"href":"https:\/\/jordan-cardiac.org\/en\/wp-json\/wp\/v2\/posts\/9227","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/jordan-cardiac.org\/en\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/jordan-cardiac.org\/en\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/jordan-cardiac.org\/en\/wp-json\/wp\/v2\/users\/145"}],"replies":[{"embeddable":true,"href":"https:\/\/jordan-cardiac.org\/en\/wp-json\/wp\/v2\/comments?post=9227"}],"version-history":[{"count":1,"href":"https:\/\/jordan-cardiac.org\/en\/wp-json\/wp\/v2\/posts\/9227\/revisions"}],"predecessor-version":[{"id":9228,"href":"https:\/\/jordan-cardiac.org\/en\/wp-json\/wp\/v2\/posts\/9227\/revisions\/9228"}],"wp:attachment":[{"href":"https:\/\/jordan-cardiac.org\/en\/wp-json\/wp\/v2\/media?parent=9227"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/jordan-cardiac.org\/en\/wp-json\/wp\/v2\/categories?post=9227"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/jordan-cardiac.org\/en\/wp-json\/wp\/v2\/tags?post=9227"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}